Review : Pathogenesis and promising non-invasive markers for preeclampsia
분야
의약학 > 산부인과학
저자
( Young Ju Kim )
발행기관
대한산부인과학회
간행물정보
Obstetrics & Gynecology Science 2013년, 제56권 제1호, 2~7페이지(총6페이지)
파일형식
40326317.pdf [무료 PDF 뷰어 다운로드]
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    영문초록
    Preeclampsia is one of the leading causes of maternal mortality/morbidity and preterm delivery in the world, affecting 3% to 5% of pregnant women. The pathophysiology of preeclampsia likely involves both maternal and fetal/placental factors. Abnormalities in the development of placental vessels early in pregnancy may result in placental hypoperfusion, hypoxia, or ischemia. Hypoperfusion, hypoxia, and ischemia are critical components in the pathogenesis of preeclampsia because the hypoperfused placenta transfers many factors into maternal vessels that alter maternal endothelial cell function and lead to the systemic symptoms of preeclampsia. There are several hypotheses to explain the pathogenesis of preeclampsia, including altered angiogenic balance, circulating angiogenic factors (such as marinobufagenin, a bufadienolide trigger), and activation of the renin-angiotensin system. Epigenetically-modified cell-free nucleic acids that circulate in plasma and serum might be novel markers with promising non-invasive clinical applications in the diagnosis of preeclampsia.
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