소개글
[의학과][신경외과] 신경외과 사례(54세 남성환자)에 대한 자료입니다.
목차
1.Physical Examination
2.Neurologic Examination
3.Initial Lab Finding
4.Summuary
5.Problem List
6.Initial Impression
7.Plan
8.Chest X-ray
9.Summary
본문내용
set of symptoms after reducing or stopping alcohol consumption
largely due to central nervous system being in a hyper-excitable state
can be fatal
direct glutamate excitotoxcity
elevated heart rate and blood pressure
electrolyte imbalance
malnutrition.
May includes seizure, delirium tremens
occurs as a result of neuro-adaptation from chronic exposure
neuropsychiatric excitability and autonomic disturbances
occurs upon declining blood levels of alcohol
can be alleviated by reintroduction of alcohol or cross-tolerant agent
Dependence on other sedative-hypnotics increases the severity
Exposure to substance causes counter-regulatory mechanism
When the substance is removed
residual counter-regulatory mechanisms withdrawal symptoms
Ethanol interacts with GABA, glutamate (NMDA), and opiates
binds to postsynaptic GABAA receptors
chloride influx, hyperpolarization of cell
Ultimately producing sedation
Long-term use of ethanol: downregulation of GABAA receptors