treatment. Integrin, EGFR are usually the target receptors.
Figure 3 A description of passive and active targeting. Blood vessel near tumor is not well organized, and numbers of pores are found. EPR effect is observed only at the tumor tissue. Through this effect, drugs are accumulated.
3. Problem and things to be advanced
We cover the meaning of cancer targeting, and how to target
3. Mechanism of Epilepsy
Under normal circumstances,
the neurotransmitter GABA does its part to make sure the system stays in Synch
by triggering signals in the form of charged particles(A)
It causes
a large concentration of Cl-
to enter the receiving neuron
This tells
the neuron to not pass on the message
treatment and fermentation process, After algae produce sugar or starch from carbon dioxide, sunlight and water, those free complex carbohydrates turned into fermentable sources through pre-treatment process - saccharification. Then, it can be changed ethanol form through fermentation by yeast or genetically engineered E.coli and we can produce biofuel from that ethanol. Now, the way of decreasin
, 전체적으로 설명하기엔 미약.
환경적 요소(diet, exercise)와 함께 영향을 줌
2. Metabolic Factors :
- 뇌의 mechanism이 set point를 유전적으로 결정짓는 부근에 몸무게를 유지하도록 하는 신체의 신진대사를 통제한다고 말한다.
- 칼로리 섭취 감소가 옛날 조상들이 흉작에 견딜 수 있도록 돕는
Mechanism & Process)
(최신) 헌팅턴병의 세포 사멸 기작 규명
- 세포사멸은 돌연변이 단백질에 의한 세포 생존에 중요한 분자 물질의 고갈 때문.
① CAG 코돈의 과다 반복에 의한 헌팅턴 유전자의 돌연변이.
② 변종의 헌팅턴 유전자가 비정상적인 헌팅턴 단백질을 발현.
③ 발현된 단백질은